File:Potential Mechanisms of Cancer-Related Hypercoagulability.png
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English: Cancer-associated thrombosis can result from: (1) stasis, i.e., direct pressure on blood vessels by the tumor mass, poor performance status, and bed rest following surgical procedures; (2) iatrogenic, due to treatment with antineoplastic medications; and (3) secretion of heparanase from malignant tumors that results in degradation of endogenous heparin. |
| Date | |
| Source | Potential Mechanisms of Cancer-Related Hypercoagulability. Nasser NJ, Fox J, Agbarya A. Cancers (Basel). 2020 Feb 29;12(3):566. https://doi.org/10.3390/cancers12030566 |
| Author | Nicola J. Nasser, Jana Fox, and Abed Agbarya |
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
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| current | 23:26, 19 February 2023 | | 1,999 × 1,135 (794 KB) | Jul059 (talk | contribs) | Lossless file size reduction |
| 06:24, 2 January 2021 |  | 1,999 × 1,135 (842 KB) | Sannecola (talk | contribs) | Uploaded a work by Nicola J. Nasser, Jana Fox, and Abed Agbarya from Potential Mechanisms of Cancer-Related Hypercoagulability. Nasser NJ, Fox J, Agbarya A. Cancers (Basel). 2020 Feb 29;12(3):566. https://doi.org/10.3390/cancers12030566 with UploadWizard |
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